پروفسور دکتر حسین نوری درمان توسط گیاهان دارویی

Expert opinion of 32 subspecialty physicians Prof. Dr. Hossein Nouri's clinic regarding SME disease Each patient after receiving a complete test (check) is sent to the admission unit for free visit is done online after examination by subspecialty physicians if they are admitted and a prescription is issued  Ghati will be made by medicinal plants, ie molecular assimilation of the patient's body with medicinal plants will be cultivated, then medicinal plants will be grown and extracted, and 100% organic, deactivated, de-fungal and virus-treated by supervising 100% of the country's health.  Due to the fact that the drug has been matched with tapeworms, the absorption of the drug has been 100%. In the field of treatment, 90 out of 100 people have improved with this method.

 Explanation about the disease (SME)
 Spinal muscular atrophy (SMA) is the breakdown or reduction in muscle size that ultimately results in decreased muscle strength and mobility.

 SMA, or spinal muscular atrophy, is a rare genetic disorder in which nerve cells in the spinal cord die prematurely, causing the muscles that normally produce those nerve cells to die.  Are controlled, suffer from degeneration.  In this disease, regardless of race or gender, one in every 10,000 babies worldwide is affected by autosomal recessive disease.
 Signs and symptoms of SMA are usually seen in the first few months after birth, which can last into adulthood, but babies bo with the most severe form of SMA often do not reach a second birth.  The disease does not affect mental abilities, but it can affect a person's ability to perform basic physical functions such as walking, breathing and swallowing.
 Diagnosis of SMA by understanding the relationship between the brain, nerves and muscles
 To understand SMA, you need to recognize the relationship between the brain, nerves, and muscles.  When the brain wants to contract a muscle, it sends a signal through the upper motor neurons that carry the message from the brain to the spinal cord, then from there through the lower motor neurons to the message from the spinal cord to the spinal nerve junction.  The lower motor neurons that cause voluntary contraction of skeletal muscle are called alpha motor neurons. The cell body these days is located in the anterior ho of the spinal cord and their axons start from the spinal cord and continue to the muscle cell.
 This condition, characterized by degeneration of motor neurons and progressive degeneration of muscle strength, is caused by a deficiency of the smn protein.  Healthy people have two genes that produce the smn protein on the q5 chromosome.
 Smn1 gene and copies of this gene called smn2.  People with SMA lack the smn1 gene and depend on one or more copies of the smn2 gene to produce the smn protein.
 The number of copies of the smn2 gene in smn patients is related to the severity of their disease, but this gene is a weak support.  Laboratory studies have shown that the smn2 gene is converted into a complete and efficient smn protein in only 10% of cases.  This is due to an error when editing RNA.
 There are four main types of the disease, each of which is diagnosed according to the severity and different effects it has on each person.  About half of the population is the first type of the disease, the symptoms of which appear before the age of six months, and this is the most severe type of SMA.  These patients have never been able to sit still, dependent on others, and are expected to live less than two years.
 In people with type 2 SMA, the onset of symptoms begins at 6 to 18 months of age and they are usually able to sit up without relying on others, but need help to stand.  Life expectancy for these patients is more than two years.  Or the progression of the disease in patients with both types of progressive vessels in the interdental muscles causes respiratory dysfunction, which if continued, may lead to respiratory failure.
 People with type 3 SMA typically experience mild motor symptoms at some point in their lives and may be able to walk independently in adulthood.  The life expectancy for these patients is comparable to that of healthy individuals.  With late onset of symptoms, progressive weakness of the legs in patients can lead to complete loss of mobility and the need to use mobility equipment. This mobility defect can lead to obesity and orthopedic diseases such as spinal curvature, all of which reduce  Mobility is associated in patients.
 Cells need protein for muscle function
 To function as muscles in the body, our cells need protein.  In a healthy person, genes are able to produce the necessary proteins at the right time and in the right amount.  One of the proteins that is very important for cell function is called smn.  In a genetic disorder called spinal muscular atrophy, cells can not produce enough healthy smn protein because of defective genes.
 In this disease, the nerve cells in which the process of producing smn proteins is not done well die, and when the patient dies, the patient loses the ability to use his muscles, so that the child with SMA loses muscle weakness one month after birth.  Encounters itself.
 Muscle weakness and weakness after childbirth can impair basic functions such as breathing, eating and sleeping.  In some clinical trials, researchers have used gene therapy to transfer the healthy smn1 gene to diseased cells.
 ZolgenSMA

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